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Biochemical basis of alcoholism : statesments and hypothesis of present research
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  • Biochemical basis of alcoholism : statesments and hypothesis of present research
Utgivning, distribution etc.
  • ANKHO International inc, Fayetteville : 1985
Fysisk beskrivning
  • S. 711-788
Anmärkning: Allmän
  • PER=Alcohol 2(1985):6
Anmärkning: Innehållsbeskrivning, sammanfattning
  • Experimental results and theoretical considerations on the biology of alcoholism are devoted to the following topics: 1. Genetically determined differences in metabolic tolerance, 2. Participation of the alternative alcohol metabolizing systems in chronic alcohol intake, 3. Genetically determined differences in functional tolerance of the CNS to the hypnotic effect of alcohol, 4. Cross tolerance between alcohol and centrally active drugs, 5. Dissociation of tolerance and cross tolerance from physical dependence, 6. Permanent effect of uncontrolled drinking behavior induced by alkaloid metabolites in the CNS, 7. Genetically determined alterations in the function of opiate receptors, and 8. Genetic predisposition to addiction due to innate endorphin deficiency.
Term
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*000      ab
*00121498
*008070120|1985|||||||||||e||||||||||0 eng|d
*1001 $aTopel, Helga
*24510$aBiochemical basis of alcoholism :$bstatesments and hypothesis of present research /$cHelga Topel
*260  $aFayetteville :$bANKHO International inc,$c1985$y1985
*300  $aS. 711-788
*500  $aPER=Alcohol 2(1985):6
*520  $aExperimental results and theoretical considerations on the biology of alcoholism are devoted to the following topics: 1. Genetically determined differences in metabolic tolerance, 2. Participation of the alternative alcohol metabolizing systems in chronic alcohol intake, 3. Genetically determined differences in functional tolerance of the CNS to the hypnotic effect of alcohol, 4. Cross tolerance between alcohol and centrally active drugs, 5. Dissociation of tolerance and cross tolerance from physical dependence, 6. Permanent effect of uncontrolled drinking behavior induced by alkaloid metabolites in the CNS, 7. Genetically determined alterations in the function of opiate receptors, and 8. Genetic predisposition to addiction due to innate endorphin deficiency.
*650 4$aAlcoholism
*650 4$aGenetics
*650 4$aEtiology
*650 4$aMetabolism
*653  $aAlkoholism
*653  $aArv
*653  $aMetabolism
*653  $aEtiologi
^
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