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Titel och upphov Brain development : normal processes and the effects of alcohol and nicotine
Utgivning, distribution etc. Oxford University Press, Oxford : 2006
Fysisk beskrivning
Anmärkning: Innehållsbeskrivning, sammanfattning Part I. Normal development . Models of neurotoxicity provide unique insight into normal development . Cell proliferation. Neuronal migration. Neuronal differentiation: from axons to synapses. Cell death. Intracellular pathways of neuronal death. Development al disorders and evolutionary expectations: mechanisms of resilience. Part II. Ethanol-affected development . Prenatal alcohol exposure and human development . Influence of alcohol on the structure of the developing human brain. Prenatal ethanol exposure and fetal programming: implications for endocrine and immune development and long-term health. Early exposure to ethanol affects the proliferation of neuronal precursors. Effects of ethanol on the regulation of cell cycle in neural stem cells. Mechanisms of ethanol-induced alterations in neuronal migration. Effects of ethanol on mechanisms regulating neuronal process outgrowth. Neuronal survival is compromised by ethanol: extracellular mediators. Intracellular events in ethanol-induced neuronal death. Neural crest and development al exposure to ethanol. Glial targets of development al exposure to ethanol. Part III. Nicotine-affected development . Tobacco use during pregnancy: epidemiology and effects on offspring. Prenatal nicotine exposure and animal behavior. Neuronal receptors for nicotine: functional diversity and development al changes. Neural precursors as preferential targets for drug abuse: long term consequences and latent susceptibility to central nervous system disorders. Nicotinic receptor regulation of developing catecholamine systems. (From the table of contents)
Term
Indexterm - Okontrollerad
Personnamn
ISBN 0-19-518313-4 978-0-19-518313-9
*000 am
*00113011
*008070120|2006||||xxk||||e||||||||1|0|eng|d
*020 $a0-19-518313-4$x0-19-518313-4
*020 $a978-0-19-518313-9$x978-0-19-518313-9
*24510$aBrain development :$bnormal processes and the effects of alcohol and nicotine /$cedited by Michael W. Miller
*260 $aOxford :$bOxford University Press,$c2006$y2006
*300 $axv, 404 s :$bill.
*520 $aPart I. Normal development . Models of neurotoxicity provide unique insight into normal development . Cell proliferation. Neuronal migration. Neuronal differentiation: from axons to synapses. Cell death. Intracellular pathways of neuronal death. Development al disorders and evolutionary expectations: mechanisms of resilience. Part II. Ethanol-affected development . Prenatal alcohol exposure and human development . Influence of alcohol on the structure of the developing human brain. Prenatal ethanol exposure and fetal programming: implications for endocrine and immune development and long-term health. Early exposure to ethanol affects the proliferation of neuronal precursors. Effects of ethanol on the regulation of cell cycle in neural stem cells. Mechanisms of ethanol-induced alterations in neuronal migration. Effects of ethanol on mechanisms regulating neuronal process outgrowth. Neuronal survival is compromised by ethanol: extracellular mediators. Intracellular events in ethanol-induced neuronal death. Neural crest and development al exposure to ethanol. Glial targets of development al exposure to ethanol. Part III. Nicotine-affected development . Tobacco use during pregnancy: epidemiology and effects on offspring. Prenatal nicotine exposure and animal behavior. Neuronal receptors for nicotine: functional diversity and development al changes. Neural precursors as preferential targets for drug abuse: long term consequences and latent susceptibility to central nervous system disorders. Nicotinic receptor regulation of developing catecholamine systems. (From the table of contents)
*650 4$aBrain
*650 4$aPregnancy
*650 4$aEthanol
*650 4$aNicotine
*650 4$aFetal development
*650 4$aToxicology
*653 $aGraviditet
*653 $aFosterutveckling
*653 $aAlkohol
*653 $aNikotin
*653 $aHjärnan
*653 $aToxikologi
*7001 $aMiller, Michael W.$4edt
*852 $h4b115
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